Medical

Plantar fasciitis and other causes of heel and sole pain

INTRODUCTION — Disorders of the ankle and foot, including ankle sprains, tendinitis, plantar fasciitis, and bursitis, usually occur in association with movement and result from trauma. In some cases, subtle skeletal structural faults (such as limb length discrepancy) predispose to these cumulative movement disorders or repetitive strain injuries. It is therefore important to conduct a careful examination to detect these defects.

Other factors can contribute to recurrent or perplexing lower limb distress. These include stress fractures that occur due to osteoporosis, inflammatory arthritis due to rheumatoid arthritis, and the spondylo arthropathies which may also present with an enthesitis (inflammation at tendon insertions into bone), painful peripheral neuropathies (eg, associated with diabetes) that may also predispose to Charcot’s joints, improper footwear, moving from a building with wood flooring to one with a concrete slab, prolonged standing, weakness and unsteadiness associated with the problems of aging, and structural disorders such as joint laxity or malalignment of the lower limbs.

Painful disorders of the foot will be reviewed here. Tendinitis of the ankle and ankle sprains are discussed separately. (See “Posterior and medial ankle tendinopathies not involving the Achilles tendon” and “Ankle sprain”.)

PLANTAR FASCIITIS — The predominant symptom of plantar fasciitis is pain in the plantar region of the foot that is worse when initiating walking. Plantar fasciitis is one of the most common causes of foot pain in adults estimated to be responsible for about one million patient visits to the doctor per year in the United States [1]. The peak incidence occurs between ages 40 and 60 years in the general population with a younger peak in runners [2,3]. It may be bilateral in up to a third of cases [2,4,5].

Heel spurs often coexist [6,7]; whether they have a causal role is unknown. They may represent a secondary response to an inflammatory reaction [2].

Anatomy — The deep plantar fascia (plantar aponeurosis) is a thick, pearly white tissue with longitudinal fibers intimately attached to the skin (figure 1). The central portion is thickest and attaches to the medial process of the tuberosity of the calcaneus; distally it divides into five slips, one for each toe. The plantar fascia provides support; as the toes extend during the stance phase of gait, it is tightened by a windlass mechanism, resulting in elevation of the longitudinal arch, inversion of the hindfoot, and a resultant external rotation of the leg [8].

Etiology — The etiology is poorly understood and is probably multifactorial. Possible risk factors for the development of plantar fasciitis include obesity, prolonged standing or jumping, flat feet, reduced ankle dorsiflexion, and heel spurs [3,5,7,9-11]. There is a high incidence in runners, suggesting that plantar fasciitis, at least in this population, is due to an injury caused by repetitive microtrauma [12]. In this group, the following have been proposed as risk factors [13-16]:

  • Excessive training (particularly a sudden increase in the distance run)
  • Faulty running shoes
  • Running on unyielding surfaces
  • Flat feet (pes planus or pronated ankles)
  • Limited ankle dorsiflexion (eg, due to a shortened Achilles tendon)
  • Pes cavus (high-arched) foot

However, evidence of an association for most of these factors is limited or absent [14].

Plantar fasciitis is common among ballet dancers [17] and those performing dance aerobic exercise. Stress applied to the Achilles tendon, either due to muscle contraction or passive stretching, results in increased tension in the plantar fascia [18]. Decreased knee extension, as may occur with tight hamstring muscles, causes an increase in loading of the forefoot when walking [19]; this could in turn increase the stress on the plantar fascia.

Plantar fasciitis usually occurs as an isolated problem but may be associated with systemic rheumatic diseases particularly reactive arthritis and the spondyloarthritides. Plantar fasciitis has been reported in association with fibromyalgia [20], fluoride used for the treatment of osteoporosis [21], and may be the presenting symptom in patients with nutritional osteomalacia [22].

Pathology — The site of abnormality is typically near the origin of the plantar fascia at the medial tuberosity of the calcaneum. Specimens of plantar fascia obtained during surgery for plantar fasciitis reveal a spectrum of changes, ranging from degeneration of the fibrous tissue, to fibroblastic proliferation, with or without evidence of chronic inflammation [23-25].

Diagnosis — A hallmark for diagnosis of plantar fasciitis is local point tenderness. This is best elicited by the examiner dorsiflexing the patient’s toes with one hand in order to pull the plantar fascia taut, and then palpating with the thumb or index finger of the other hand along the fascia from the heel to the forefoot (picture 1). Points of discrete tenderness can be found and marked for possible later injection.

Laboratory testing is not helpful in the diagnosis of plantar fasciitis. Tests for inflammation (eg, erythrocyte sedimentation rate and C-reactive protein) will be normal unless there is coexistent inflammatory disease.

Radiographic studies may be required to establish the diagnosis when this is in doubt in patients with persistent plantar pain. The primary goal of radiography is to rule out other disorders, especially calcaneal stress fractures. Lateral and axial radiographs are the appropriate views due to the myriad of diagnostic possibilities. The presence of heel spurs is of no diagnostic value in either ruling in or ruling out plantar fasciitis. In one study, 85 percent of 27 patients with plantar fasciitis and 46 percent of 79 controls had calcaneal spurs detected on plain non-weight bearing lateral X-Rays read by a radiologist blinded to the clinical diagnosis [26]. On the other hand, increased plantar fascia thickness and fat pad abnormalities detected in the same X-Rays had a sensitivity of 85 percent and specificity of 95 percent for plantar fasciitis.

The soft tissue may be evaluated with magnetic resonance imaging (MRI) in cases resistant to treatment [27]. Features suggestive of plantar fasciitis are thickening of the plantar fascia and increased signal on delayed (T2) and short tau inversion recovery (STIR) images [28]. Technetium scintigraphy has also been successful in localizing the inflammatory focus and ruling out stress fracture [29].

Ultrasonography of the foot may also be useful in detecting plantar fascial thickening, hypoechogenicity at the insertion upon the calcaneus, blurring of the boundary between fascia and surrounding tissues, and decreased echogenicity suggestive of edema [30,31]. The sensitivity and specificity of ultrasonography for the diagnosis of plantar fasciitis was 80 percent and 88.5 percent respectively in a study of 77 patients and a similar number of asymptomatic controls [32]. Doppler ultrasound may improve the value of this technique and provide additional information on local hyperemia [33].

Differential diagnosis — Plantar heel pain and pain in the sole of the foot may be induced by a number of other disorders. These include the following [34-36]:

  • Rupture of the plantar fascia — Rupture of the plantar fascia generally follows physical activity and has a sudden onset, unlike the more gradual appearance of the pain of plantar fasciitis. Examination of the affected foot may reveal a loss of height of the arch and there may be visible swelling or ecchymosis present.
  • Nerve pain due to entrapment — Entrapment of the posterior tibial nerve as it courses beneath the medial malleolus can cause pain, paresthesia, and numbness on the sole of the foot. Percussion tenderness over the posterior tibial nerve in the tarsal tunnel is characteristically found. (See “Overview of lower extremity peripheral nerve syndromes”, section on ‘Tarsal tunnel syndrome’.)
  • Compression or trauma to branches of the posterior tibial nerve, particularly the medial calcaneal branch or to the nerve supplying the abductor digiti quinti can also cause pain in the heel.
  • Bone pain — Pain arising from the calcaneus may also be associated with exacerbation during weight bearing. Pain caused by a neoplasm or infection is typically constant or characterized by nocturnal worsening. Stress fractures should be considered if there has been an increase in physical activity. While plain radiographs may be diagnostic, early changes with infection, tumor, and stress fracture are better differentiated by MRI. (See “Overview of cancer pain syndromes” and “Overview of osteomyelitis in adults”, section on ‘Diagnosis’ and “Overview of stress fractures”.)

    Another cause of calcaneal bone pain is Paget disease, which is generally apparent on plain radiographs with coarsened trabecular bone and may be accompanied by an elevated serum alkaline phosphatase. (See “Clinical manifestations and diagnosis of Paget disease of bone”.)

  • Tendinitis of the posterior tibialis or flexor digitorum longus tendons — These typically have an insidious onset with pain and tenderness along the course of the tendons and tendon sheaths. Subtle changes in the position of bones of the midfoot may be indicative of tendon rupture. (See “Posterior and medial ankle tendinopathies not involving the Achilles tendon”.)
  • Reactive arthritis and other spondyloarthritides — Asymmetric involvement and a propensity for the joints and entheses of the lower extremities are frequently seen in the spondyloarthritides. Back pain with inflammatory features (eg, night pain, prolonged morning stiffness) is frequently present in patients with ankylosing spondylitis, while a prior history of enterocolitis or genitourinary infection is suggestive of reactive arthritis. (See “Reactive arthritis (formerly Reiter syndrome)” and ‘Calcaneal periostitis’ below.)
  • Painful heel pad syndrome — The painful heel pad syndrome most often occurs in marathon runners. It is thought to result from disruption of the fibrous septae that compartmentalize the fat in the heel pad. Pain is localized to the heel pad; the plantar fascia is not tender and pain is not accentuated as the examiner dorsiflexes the toes. Insertion of heel cups [37] and “plastizote” that is individually molded to the patient’s heel may be useful [38].
  • Atrophy of the heel pad — Atrophy of the heel pad occurs in the elderly. Palpation reveals bony prominence without the padding usually afforded by subcutaneous fat. Unlike pain due to plantar fasciitis, pain due to atrophy of the heel pad is absent in the morning and develops and worsens during weight bearing throughout the day.
  • Piezogenic papules — Piezogenic papules are herniations of fat that occur as painful papules at the medial inferior border of the heel. They may be noted only upon weightbearing and are an uncommon cause of painful heels. Weight reduction, use of felt padding, and cushion-soled or crepe-soled shoes may provide relief [39].
  • Sarcoidosis — Heel pain has been reported to occur in sarcoidosis [40]. The combination of erythema nodosum, hilar adenopathy, migratory polyarthralgias, and fever is referred to as Lofgren’s syndrome, and is typically seen in patients with sarcoidosis. (See “Erythema nodosum”.)

Treatment

Conservative therapy — Treatment of obesity, symptomatic flat feet, and systemic inflammation should be undertaken when these conditions are present. Otherwise, treatment should begin with conservative therapy including measures to relieve pain, alterations in their shoes or habits, and exercise therapy. It should be noted there are limited data for the effectiveness of most of these modalities in the treatment of plantar fasciitis [34,41].

  • Rest and icing may give initial pain relief.
  • Nonsteroidal antiinflammatory drugs (NSAIDs) are often used. A well-designed but small trial that randomly assigned 29 patients to NSAID or placebo reported a nonsignificant trend toward improved pain and disability in the NSAID group [42]. Use for longer than two or three weeks should be reserved for patients with systemic inflammation.
  • There are conflicting reports on the benefit of resting padded foot splints [43-46]; these splints can usually be purchased in pharmacies that feature orthopedic supplies (picture 2). The splints are worn at night to keep the ankle in the neutral position with or without dorsiflexion of the metatarsophalangeal joints during sleep. A clinical trial reported they were of similar effectiveness to custom-fitted orthotics (see below) although there was better compliance and fewer side effects reported with orthoses use [47].
  • Prefabricated silicone heel inserts combined with stretching exercises (see below) may be of value [48]. Felt pads or rubber heel cups appear to be less effective than silicone inserts; magnetic insoles have not been found to provide additional benefit compared with nonmagnetic insoles [49,50].
  • Wearing slippers or going barefoot may aggravate symptoms or result in a recurrence of symptoms. Thus, the first step out of bed should be made with a supportive shoe or sandal on.
  • Patients who work or reside in buildings with concrete floors should use cushion-soled or crepe-soled shoes. Excessive heel impact from jumping, excessive heel impact during walking, or use of a trampoline should be avoided.
  • Athletic shoes, arch supporting shoes (particularly those with an extra-long counter, which is the firm part of the shoe that surrounds the heel), or shoes with rigid shanks (usually a metal insert into the sole of the shoe) may be helpful. Shoes with these features can be found in stores featuring work shoes or “orthopedic shoes”.
  • Exercises may be beneficial although evidence is limited. Home exercises include the calf-plantar fascia stretch (picture 3), foot/ankle circles (picture 4), toe curls (picture 5), and toe towel curls (picture 6). One unblinded trial noted that non-weight bearing stretching exercises specific to the plantar fascia were more beneficial in the short term than weight-bearing Achilles tendon-stretching exercises [51], although the method of analysis may have biased towards this result. At two-year follow up there were no differences between groups [52]. Another placebo-controlled trial found no significant differences in pain and function in those who undertook a two-week calf muscle stretching program compared to those who did not [53]. Ultrasound therapy, ice massage, and deep friction massage may be used prior to exercise although their effectiveness is unknown.
  • Tape support of the affected plantar surface, a technique referred to as low-Dye taping may be beneficial to some patients particularly for first-step pain [54,55]. Four strips of tape are applied as illustrated in the figure (figure 2). The tape should not be applied too tightly and use of hypoallergenic tape is recommended to avoid allergic reactions [55].

If these inexpensive and noninvasive measures fail to bring about improvement within two to three weeks, the following may be considered:

  • The points of tenderness along the plantar fascia may be injected with a glucocorticoid/local anesthetic mixture (picture 7). Injections may provide short-term, temporary pain relief. In a randomized trial involving 106 patients, significantly less pain was noted in patients who received a single injection of 1 mL each of glucocorticoid and local anesthetic (lidocaine) than with an injection of 2 mL of local anesthetic alone, a difference that was significant at one month, but not at three or six months [56]. In one non-randomized trial in which four types of injections were compared (25 consecutive patients in each group), those who received glucocorticoid (2 mL of triamcinolone) either as a single injection or using a peppering technique were reported to have better outcomes than those who received either 2 mL autologous blood or 2 mL lidocaine using a peppering technique [57].

    An alternative, possibly less painful approach, a medial injection, may also provide significant benefit [58]. One of the authors (RS) injects into a minimum of three tender locations along the fascia while the other author (RB) uses a single injection directed to the site of maximal tenderness. The utility of using ultrasound to guide placement of the injection remains uncertain. Two randomized trials have found pain relief from ultrasound-guided injection was no different to blind injection [59,60] although one trial reported a lower recurrence rate [60].

    Glucocorticoid injection should be used judiciously since repeated injection may cause heel pad atrophy [61]. It may also predispose to plantar fascia rupture [62,63] although evidence for this is limited and non-conclusive. One study reported a series of 37 patients with a presumptive diagnosis of plantar fascia rupture, all of whom had had a prior episode of plantar fasciitis treated with glucocorticoid injection into the calcaneal origin of the fascia [62]. In another study of 765 patients with plantar fasciitis, 43 of 51 patients with plantar fascia rupture had received one or more glucocorticoid injections although the number of patients without plantar fascial rupture who received one or more glucocorticoid injections was not reported [63].

    A randomized trial of glucocorticoid injection compared with extracorporeal shock wave therapy (see ‘Shock wave therapy’ below) in 132 patients found that after three months there was significantly less pain in those who received the injection (mean differences in visual analog pain of 1.48 versus 3.69, respectively) [64].

    Injection of autologous blood has been proposed as a treatment for plantar fasciitis on the basis that it may begin a cascade of local factors to stimulate angiogenesis and healing [A6]. While no trials have assessed the efficacy of autologous blood injection compared with placebo for plantar fasciitis, two trials have compared this treatment to glucocorticoid injection [57,65]. While participants improved over time irrespective of treatment received, both trials reported significant differences in benefit favoring the glucocorticoid groups [57,65].

  • Custom-fabricated inserts, usually provided by podiatrists, include inserted orthoses with foam rubber raised arches and rubber or tub heels, and molded ankle-foot orthoses. A meta-analysis that included four trials and two prospective cohort studies concluded that the use of orthoses and night splints results in significant reductions in self-reported pain, but only orthoses significantly improve function [66]. A recent Cochrane systematic review that identified five trials including 691 participants that assessed the use of custom-made foot orthoses for plantar fasciitis was inconclusive [67].

    The efficacy of foot orthoses remains controversial, and there are considerable variations in the prescribing habits of podiatrists, orthopedists, and prosthetists [68-70]. Any potential advantage of customized orthotics over prefabricated ones must be weighed against a substantial difference in cost, estimated to be approximately two to six-fold more for the customized version [69]. A short walking cast is used by some orthopedists although again there are no published trials of this treatment approach.

  • Iontophoresis with 0.4 percent dexamethasone (six sessions over two weeks) provided moderate initial relief of plantar pain in a small, randomized placebo-controlled trial in runners with plantar fasciitis although this effect was not maintained at 4 weeks [71]. In another small study, low-Dye taping combined with iontophoresis with a 5 percent solution of acetic acid was superior to taping and iontophoresis with 0.4 percent dexamethasone but no different to taping and iontophoresis with placebo [72].

Shock wave therapy — The effectiveness of extracorporeal shock wave therapy for plantar fasciitis has been more extensively studied than any other single treatment modality. As of April 2007, at least 14 randomized controlled trials that have compared shock wave therapy with either placebo or sub-therapeutic doses of shock waves have been published in English language journals [73-75]. These trials have been of variable methodological quality and have reported conflicting results. A systematic review published in 2005 included 11 trials and performed a pooled analysis of data from 6 trials involving 897 patients [73]. The authors concluded there was no clinically important benefit of shock wave therapy despite a small statistically significant benefit in morning pain of less than 0.5 cm on a 10 cm visual analogue scale. No statistically significant benefit was observed in a sensitivity analysis that only included high-quality trials. A recent trial of a new pneumatic low-energy extracorporeal shock wave device also reported that outcomes from shock wave treatment were no better than sham therapy in a trial of 25 participants [76].

There is ongoing clinical uncertainty about the effectiveness of shock wave therapy; opinions are highly polarized, fuelled by the lack of convergence of findings from randomized evaluations. Explanations that have been put forward to explain the differing results include variation in methodological quality, the different types of equipment that have been used to generate the shock waves, different delivery methods and different doses. Duration of symptoms has not been found to influence response to treatment [77,78]. An association between therapeutic response and the amount of the shock wave energy applied was noted in one study [75]. The major reported adverse effect is transient pain at the time of treatment.

Botulinum toxin injection — A single small double-blind placebo-controlled trial in 27 patients with unilateral or bilateral plantar fasciitis assessed the effect of two injections of botulinum toxin (40 units); one into the foot near the calcaneal tuberosity and the other in the arch (30 units) [79]. Feet injected with botulinum toxin had more improvement in pain and tenderness at 3 and 8 weeks than those injected with saline solution (placebo) although effect sizes were not reported. Further trials are needed before considering this treatment approach for usual care.

Complementary therapies — Topical application of wheatgrass cream twice daily for 6 weeks was ineffective in a recent randomized double-blind placebo-controlled trial involving 80 participants [80]. Other trials of complementary therapies are expected.

Radiotherapy — Radiation therapy is sometimes used in Europe to treat chronic plantar fasciitis that is unresponsive to more conservative approaches [81]. Its effectiveness has not been assessed in randomized controlled trials and whether there is a long term increased risk of carcinogenesis is unknown. Because of concern about the possibility of late onset hematopoietic malignancy, radiation therapy is seldom used to treat plantar fasciitis in other parts of the world.

A survey of German radiation treatment institutions found that 81 percent provided radiation therapy for heel pain associated with calcaneal spurs [81]. There was wide variation in the amount of radiation delivered per session (median 1 Gy) and the total amount of radiation (median 6 Gy). Retrospective review of the outcomes of a single course of therapy in 7947 patients at 76 centers found that complete pain relief lasting at least 3 months occurred in 25 to 100 percent of patients (median 70 percent) and was not related to the dose of radiation delivered. Approximately 15 percent of treatment courses resulted in no pain relief.

Surgical therapy — It is estimated that 2 to 5 percent of patients with plantar fasciitis undergo surgical procedures [2,4,12,82] although the rate may be much lower. As an example, a report of the surgical experience at the Mayo Clinic found that only 16 operations had been performed during a twelve year study period [83]. Combining release of the plantar fascia and the first branch of the lateral plantar nerve may enhance results. In one series of 28 patients (33 feet), 90 percent had a good outcome and would recommend the procedure to others [84].

While numerous surgical procedures have been described, none have been assessed in controlled trials. Favorable outcomes are reported in more than 75 percent of published case series although recovery time may be prolonged and persistent pain is not uncommon. Variations of open or closed partial of complete plantar fascia release with or without calcaneal spur resection, excision of abnormal tissue and nerve decompression have been described. Exostoses (bony projections) in runners and dancers may also improve with surgery [85].

In comparison to open release, closed procedures may allow more rapid recovery and resumption of usual activities [86,87]. In an uncontrolled series of 16 runners and 10 walkers with refractory plantar fasciitis, uniportal plantar fasciotomy gave good or excellent results provided the patient’s body mass index was less than 27 [88]. Runners in this series required a mean of 2.6 months before returning to jogging.

Potential complications of surgery include transient heel pad swelling, calcaneal fracture injury to the posterior tibial nerve or its branches and flattening of the longitudinal arch with resultant midtarsal pain.

Cryosurgery — Promising results have recently been observed in a single study that used percutaneous cryosurgery, a minimally invasive technique for freezing tissue, to treat plantar fasciitis in 59 patients [89]. The effectiveness of this technique needs to be assessed in controlled trials.

Prevention — The efficacy of preventive strategies such as stretching exercises and controlling the intensity of running (eg distance, frequency and duration) in specifically preventing plantar fasciitis is unknown [90]. Footwear designed to maximize shock absorption may be of value [91].

Prognosis — The outcome for patients with plantar fasciitis is generally favorable; approximately 80 percent of patients have complete resolution of pain within one year [4,82,92,93]. The favorable natural history of this benign condition should be borne in mind when weighing the potential benefits and risks of unproven and sometimes costly treatments.

Summary and recommendations — Plantar fasciitis is a painful disorder that is associated with degenerative and sometimes chronic inflammatory changes in the affected tissue. It has a good prognosis and patients can be informed that with conservative treatment 80 percent have complete resolution within one year. Although many types of treatments have been used for this disorder, few have been rigorously validated in clinical trials. Based upon the data presented earlier in this review, the following initial conservative interventions are recommended (see ‘Conservative therapy’ above):

  • Stretching exercises for the calf muscles and the plantar fascia, which the patient can perform at home
  • Avoiding the use of flat shoes and bare-foot walking
  • Using prefabricated, over-the-counter, silicone heel shoe inserts (arch supports and/or heel cups)
  • Decreasing physical activities that are suggested by the medical history to be causative or aggravating (eg, excessive running, dancing, or jumping)
  • Prescribing or recommending a short-term (two to three weeks) trial of NSAIDs is reasonable, but long-term use should be reserved for patients with known systemic rheumatic disease
  • Injecting the tender areas of the plantar region with glucocorticoids and a local anesthetic.

If the treatments above have not produced sufficient improvement, more costly therapies can be considered although these remain unproven:

  • Molded shoe inserts (orthotics)
  • Night splints
  • Immobilization with a cast
  • Extracorporeal shock wave therapy

Surgery is generally reserved for those patients who do not respond to at least 6 to 12 months of conservative therapy.

OTHER CAUSES OF HEEL PAIN — A variety of other disorders may cause heel pain.

Haglund’s syndrome — A prominent enlarged bony posterior superior calcaneal tubercle sometimes causes compression of soft tissue and foot pain (which may be due in part to a retrocalcaneal bursitis) in the posterior area of the heel over the bony prominence, above the site of attachment of the Achilles tendon [94]. The cause is usually a combination of developmental problems and altered foot dynamics including a more vertical pitch of the posterior portion of the calcaneus.

Patients with Haglund’s syndrome (also called posterior calcaneal tubercle impingement syndrome) come to the physician due to an inability to wear shoes comfortably. A 2 to 3 mm bony extrusion is visible and palpable. The overlying skin may contain an area of erythema and swelling.

Nonoperative treatment of Haglund’s syndrome includes a well-fitted heel cup, a laced or strapped shoe that reduces heel counter friction, and heel padding to raise the heel. A “V” cutout heel counter to decompress the bursa may be used if bursitis is present and persistent; incising the heel counter with a deep “V” will provide less friction. Surgical excision should be considered if the exostosis is large and symptoms persist [95]. Although calcaneal ostectomy provided good to excellent results in a series of 39 feet that were operated upon, six of 36 patients said they would not recommend the surgery to others, mainly due to the prolonged time to recover from the operation [96].

Calcaneal periostitis — Calcaneal periostitis may result from trauma, reactive arthritis, ankylosing spondylitis, psoriatic arthritis, or rheumatoid arthritis [97]. Pain is usually bilateral, along the lateral and plantar aspect of the heels, worse in the morning upon arising, and often accompanied by morning stiffness. Tenderness is diffusely present along the plantar aspect of the heel and midfoot, and along the lateral border of the heels.

Bilateral involvement should alert to the possibility of a coexistent systemic rheumatic disease. Fluffy calcific deposition of the plantar aspect of the heel on a lateral radiograph is suggestive of reactive arthritis.

Management includes use of NSAIDs for pain and inflammation; the resulting painful heel may have to be raised one to two cm with a heel lift for relief. Treatment of the underlying disease process will often be helpful.

Calcaneal spurs — Calcaneal spurs can develop on the plantar tuberosity and extend across the entire width of the calcaneus. The apex of the spur is embedded in the plantar fascia. Heel spurs are typically asymptomatic (picture 8); pain occurs when the apex is angled downward by depression of the long arch. An acutely painful heel spur may also be seen in certain systemic diseases, such as ankylosing spondylitis, reactive arthritis, or rheumatoid arthritis.

Conservative treatment includes a cutout heel pad or a custom-made orthotic. Surgery usually is needed if the prominence is palpable with only a thin amount of natural heel pad intervening between the spur and skin. MRI may be useful if surgery is being considered; it may delineate other factors responsible for the pain, particularly if the spur is not palpable and the diagnosis is in doubt.

INFORMATION FOR PATIENTS — UpToDate offers two types of patient education materials, “The Basics” and “Beyond the Basics.” The Basics patient education pieces are written in plain language, at the 5th to 6th grade reading level, and they answer the four or five key questions a patient might have about a given condition. These articles are best for patients who want a general overview and who prefer short, easy-to-read materials. Beyond the Basics patient education pieces are longer, more sophisticated, and more detailed. These articles are written at the 10th to 12th grade reading level and are best for patients who want in-depth information and are comfortable with some medical jargon.

Here are the patient education articles that are relevant to this topic. We encourage you to print or e-mail these topics to your patients. (You can also locate patient education articles on a variety of subjects by searching on “patient info” and the keyword(s) of interest.)

  • Basics topics (see “Patient information: Heel pain (caused by plantar fasciitis) (The Basics)”)
  • Beyond the Basics topics (see “Patient information: Heel pain (caused by plantar fasciitis)”)
Use of UpToDate is subject to the Subscription and License Agreement.

REFERENCES

  1. Riddle DL, Schappert SM. Volume of ambulatory care visits and patterns of care for patients diagnosed with plantar fasciitis: a national study of medical doctors. Foot Ankle Int 2004; 25:303.
  2. Furey JG. Plantar fasciitis. The painful heel syndrome. J Bone Joint Surg Am 1975; 57:672.
  3. Taunton JE, Ryan MB, Clement DB, et al. A retrospective case-control analysis of 2002 running injuries. Br J Sports Med 2002; 36:95.
  4. LAPIDUS PW, GUIDOTTI FP. PAINFUL HEEL: REPORT OF 323 PATIENTS WITH 364 PAINFUL HEELS. Clin Orthop Relat Res 1965; 39:178.
  5. Sadat-Ali M. Plantar fasciitis/calcaneal spur among security forces personnel. Mil Med 1998; 163:56.
  6. Onuba O, Ireland J. Plantar fasciitis. Ital J Orthop Traumatol 1986; 12:533.
  7. Prichasuk S, Subhadrabandhu T. The relationship of pes planus and calcaneal spur to plantar heel pain. Clin Orthop Relat Res 1994; :192.
  8. Greene, WB (Ed). Essentials of Musculoskeletal Care, 2nd ed, American Academy of Orthopaedic Surgeons, Rosemont, Illinois 2001. p. 487.
  9. Riddle DL, Pulisic M, Pidcoe P, Johnson RE. Risk factors for Plantar fasciitis: a matched case-control study. J Bone Joint Surg Am 2003; 85-A:872.
  10. Gill LH, Kiebzak GM. Outcome of nonsurgical treatment for plantar fasciitis. Foot Ankle Int 1996; 17:527.
  11. Rano JA, Fallat LM, Savoy-Moore RT. Correlation of heel pain with body mass index and other characteristics of heel pain. J Foot Ankle Surg 2001; 40:351.
  12. McBryde AM Jr. Plantar fasciitis. Instr Course Lect 1984; 33:278.
  13. Warren BL, Jones CJ. Predicting plantar fasciitis in runners. Med Sci Sports Exerc 1987; 19:71.
  14. Rome, K. Anthropometric and biomechanical risk factors in the development of plantar heel pain – a review of the literature. Phys Ther Rev 1997; 2:123.
  15. Messier SP, Pittala KA. Etiologic factors associated with selected running injuries. Med Sci Sports Exerc 1988; 20:501.
  16. Warren BL. Anatomical factors associated with predicting plantar fasciitis in long-distance runners. Med Sci Sports Exerc 1984; 16:60.
  17. Oloff LM, Schulhofer SD. Flexor hallucis longus dysfunction. J Foot Ankle Surg 1998; 37:101.
  18. Cheung JT, Zhang M, An KN. Effect of Achilles tendon loading on plantar fascia tension in the standing foot. Clin Biomech (Bristol, Avon) 2006; 21:194.
  19. Harty J, Soffe K, O’Toole G, Stephens MM. The role of hamstring tightness in plantar fasciitis. Foot Ankle Int 2005; 26:1089.
  20. Harvey CK. Fibromyalgia. Part II. Prevalence in the podiatric patient population. J Am Podiatr Med Assoc 1993; 83:416.
  21. Riggs BL, Hodgson SF, Hoffman DL, et al. Treatment of primary osteoporosis with fluoride and calcium. Clinical tolerance and fracture occurrence. JAMA 1980; 243:446.
  22. Paice EW, Hoffbrand BI. Nutritional osteomalacia presenting with plantar fasciitis. J Bone Joint Surg Br 1987; 69:38.
  23. Jarde O, Diebold P, Havet E, et al. Degenerative lesions of the plantar fascia: surgical treatment by fasciectomy and excision of the heel spur. A report on 38 cases. Acta Orthop Belg 2003; 69:267.
  24. Leach RE, Seavey MS, Salter DK. Results of surgery in athletes with plantar fasciitis. Foot Ankle 1986; 7:156.
  25. Lemont H, Ammirati KM, Usen N. Plantar fasciitis: a degenerative process (fasciosis) without inflammation. J Am Podiatr Med Assoc 2003; 93:234.
  26. Osborne HR, Breidahl WH, Allison GT. Critical differences in lateral X-rays with and without a diagnosis of plantar fasciitis. J Sci Med Sport 2006; 9:231.
  27. Helie O, Dubayle P, Boyer B, Pharaboz C. [Magnetic resonance imaging of lesions of the superficial plantar fasciitis]. J Radiol 1995; 76:37.
  28. McGonagle D, Marzo-Ortega H, O’Connor P, et al. The role of biomechanical factors and HLA-B27 in magnetic resonance imaging-determined bone changes in plantar fascia enthesopathy. Arthritis Rheum 2002; 46:489.
  29. Dasgupta B, Bowles J. Scintigraphic localisation of steroid injection site in plantar fasciitis. Lancet 1995; 346:1400.
  30. Gibbon WW, Long G. Ultrasound of the plantar aponeurosis (fascia). Skeletal Radiol 1999; 28:21.
  31. Groshar, D, Alperson, M, Toubi, A, et al. Plantar fasciitis: detection with ultrasonography versus bone scintigraphy. The Foot 2000; 10:164.
  32. Sabir N, Demirlenk S, Yagci B, et al. Clinical utility of sonography in diagnosing plantar fasciitis. J Ultrasound Med 2005; 24:1041.
  33. Walther M, Radke S, Kirschner S, et al. Power Doppler findings in plantar fasciitis. Ultrasound Med Biol 2004; 30:435.
  34. Buchbinder R. Clinical practice. Plantar fasciitis. N Engl J Med 2004; 350:2159.
  35. Cole C, Seto C, Gazewood J. Plantar fasciitis: evidence-based review of diagnosis and therapy. Am Fam Physician 2005; 72:2237.
  36. Singh D, Angel J, Bentley G, Trevino SG. Fortnightly review. Plantar fasciitis. BMJ 1997; 315:172.
  37. Katoh Y, Chao EY, Morrey BF, Laughman RK. Objective technique for evaluating painful heel syndrome and its treatment. Foot Ankle 1983; 3:227.
  38. Spiegl, PV, Johnson, KA. Heel pain syndrome: Which treatments to choose? J Musculoskel Med 1984; 1:66.
  39. Shelley WB, Rawnsley HM. Painful feet due to herniation of fat. JAMA 1968; 205:308.
  40. Shaw RA, Holt PA, Stevens MB. Heel pain in sarcoidosis. Ann Intern Med 1988; 109:675.
  41. Crawford F, Thomson C. Interventions for treating plantar heel pain. Cochrane Database Syst Rev 2003; :CD000416.
  42. Donley BG, Moore T, Sferra J, et al. The efficacy of oral nonsteroidal anti-inflammatory medication (NSAID) in the treatment of plantar fasciitis: a randomized, prospective, placebo-controlled study. Foot Ankle Int 2007; 28:20.
  43. Powell M, Post WR, Keener J, Wearden S. Effective treatment of chronic plantar fasciitis with dorsiflexion night splints: a crossover prospective randomized outcome study. Foot Ankle Int 1998; 19:10.
  44. Probe RA, Baca M, Adams R, Preece C. Night splint treatment for plantar fasciitis. A prospective randomized study. Clin Orthop Relat Res 1999; :190.
  45. Ryan J. Use of posterior night splints in the treatment of plantar fasciitis. Am Fam Physician 1995; 52:891.
  46. Wapner KL, Sharkey PF. The use of night splints for treatment of recalcitrant plantar fasciitis. Foot Ankle 1991; 12:135.
  47. Roos E, Engström M, Söderberg B. Foot orthoses for the treatment of plantar fasciitis. Foot Ankle Int 2006; 27:606.
  48. Pfeffer G, Bacchetti P, Deland J, et al. Comparison of custom and prefabricated orthoses in the initial treatment of proximal plantar fasciitis. Foot Ankle Int 1999; 20:214.
  49. Caselli MA, Clark N, Lazarus S, et al. Evaluation of magnetic foil and PPT Insoles in the treatment of heel pain. J Am Podiatr Med Assoc 1997; 87:11.
  50. Winemiller MH, Billow RG, Laskowski ER, Harmsen WS. Effect of magnetic vs sham-magnetic insoles on plantar heel pain: a randomized controlled trial. JAMA 2003; 290:1474.
  51. DiGiovanni BF, Nawoczenski DA, Lintal ME, et al. Tissue-specific plantar fascia-stretching exercise enhances outcomes in patients with chronic heel pain. A prospective, randomized study. J Bone Joint Surg Am 2003; 85-A:1270.
  52. Digiovanni BF, Nawoczenski DA, Malay DP, et al. Plantar fascia-specific stretching exercise improves outcomes in patients with chronic plantar fasciitis. A prospective clinical trial with two-year follow-up. J Bone Joint Surg Am 2006; 88:1775.
  53. Radford JA, Landorf KB, Buchbinder R, Cook C. Effectiveness of calf muscle stretching for the short-term treatment of plantar heel pain: a randomised trial. BMC Musculoskelet Disord 2007; 8:36.
  54. Landorf KB, Radford JA, Keenan AM, Redmond AC. Effectiveness of low-Dye taping for the short-term management of plantar fasciitis. J Am Podiatr Med Assoc 2005; 95:525.
  55. Radford JA, Landorf KB, Buchbinder R, Cook C. Effectiveness of low-Dye taping for the short-term treatment of plantar heel pain: a randomised trial. BMC Musculoskelet Disord 2006; 7:64.
  56. Crawford F, Atkins D, Young P, Edwards J. Steroid injection for heel pain: evidence of short-term effectiveness. A randomized controlled trial. Rheumatology (Oxford) 1999; 38:974.
  57. Kalaci A, Cakici H, Hapa O, et al. Treatment of plantar fasciitis using four different local injection modalities: a randomized prospective clinical trial. J Am Podiatr Med Assoc 2009; 99:108.
  58. Kamel M, Kotob H. High frequency ultrasonographic findings in plantar fasciitis and assessment of local steroid injection. J Rheumatol 2000; 27:2139.
  59. Kane D, Greaney T, Bresnihan B, et al. Ultrasound guided injection of recalcitrant plantar fasciitis. Ann Rheum Dis 1998; 57:383.
  60. Tsai WC, Wang CL, Tang FT, et al. Treatment of proximal plantar fasciitis with ultrasound-guided steroid injection. Arch Phys Med Rehabil 2000; 81:1416.
  61. D’Ambrosia RD. Conservative management of metatarsal and heel pain in the adult foot. Orthopedics 1987; 10:137.
  62. Sellman JR. Plantar fascia rupture associated with corticosteroid injection. Foot Ankle Int 1994; 15:376.
  63. Acevedo JI, Beskin JL. Complications of plantar fascia rupture associated with corticosteroid injection. Foot Ankle Int 1998; 19:91.
  64. Porter MD, Shadbolt B. Intralesional corticosteroid injection versus extracorporeal shock wave therapy for plantar fasciopathy. Clin J Sport Med 2005; 15:119.
  65. Lee TG, Ahmad TS. Intralesional autologous blood injection compared to corticosteroid injection for treatment of chronic plantar fasciitis. A prospective, randomized, controlled trial. Foot Ankle Int 2007; 28:984.
  66. Lee SY, McKeon P, Hertel J. Does the use of orthoses improve self-reported pain and function measures in patients with plantar fasciitis? A meta-analysis. Phys Ther Sport 2009; 10:12.
  67. Hawke F, Burns J, Radford JA, du Toit V. Custom-made foot orthoses for the treatment of foot pain. Cochrane Database Syst Rev 2008; :CD006801.
  68. Bordelon, RL. Practical guide to foot orthoses. J Musculoskel Med 1989; 6:71.
  69. Landorf KB, Keenan AM, Herbert RD. Effectiveness of foot orthoses to treat plantar fasciitis: a randomized trial. Arch Intern Med 2006; 166:1305.
  70. Tooms RE, Griffin JW, Green S, Cagle K. Effect of viscoelastic insoles on pain. Orthopedics 1987; 10:1143.
  71. Gudeman SD, Eisele SA, Heidt RS Jr, et al. Treatment of plantar fasciitis by iontophoresis of 0.4% dexamethasone. A randomized, double-blind, placebo-controlled study. Am J Sports Med 1997; 25:312.
  72. Osborne HR, Allison GT. Treatment of plantar fasciitis by LowDye taping and iontophoresis: short term results of a double blinded, randomised, placebo controlled clinical trial of dexamethasone and acetic acid. Br J Sports Med 2006; 40:545.
  73. Thomson CE, Crawford F, Murray GD. The effectiveness of extra corporeal shock wave therapy for plantar heel pain: a systematic review and meta-analysis. BMC Musculoskelet Disord 2005; 6:19.
  74. Kudo P, Dainty K, Clarfield M, et al. Randomized, placebo-controlled, double-blind clinical trial evaluating the treatment of plantar fasciitis with an extracoporeal shockwave therapy (ESWT) device: a North American confirmatory study. J Orthop Res 2006; 24:115.
  75. Malay DS, Pressman MM, Assili A, et al. Extracorporeal shockwave therapy versus placebo for the treatment of chronic proximal plantar fasciitis: results of a randomized, placebo-controlled, double-blinded, multicenter intervention trial. J Foot Ankle Surg 2006; 45:196.
  76. Marks W, Jackiewicz A, Witkowski Z, et al. Extracorporeal shock-wave therapy (ESWT) with a new-generation pneumatic device in the treatment of heel pain. A double blind randomised controlled trial. Acta Orthop Belg 2008; 74:98.
SEE MORE:  Fewer U.S. teens are giving birth, CDC finds


Shein.com INT

June 2016
M T W T F S S
« May   Sep »
 12345
6789101112
13141516171819
20212223242526
27282930  


tittygram INT


Aviasales.ru